By Erich F. Schmidt
The 3rd and ultimate quantity is associated with the previous volumes often by means of the photo and descriptive documentation of the tombs of the Achaemenid monarhs at Naqsh-i Rustam, and Persepolis, through a learn of the excavated Achaemenid Tower at Naqsh-i Rastum, and by way of an appended evaluation of tribute bearers and throne-bearers depicted within the Persepolis reliefs.
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Extra info for Persepolis III: The Royal Tombs and Other Monuments
The T1 tetramer most likely remains in mature Kv channels on the cell membrane (41). Does the T1 domain merely provide a physical platform for channel assembly or could it also be a receptor for regulatory molecules and be somehow involved in channel gating? 2, reveal that channel inhibition by the m1 muscarinic acetylcholine receptor is due to tyrosine phosphorylation of the channel (42). 2 channel is modulated by a small GTPase and tyrosine phosphatase that physically interact with its N-terminal T1 domain (43, 44), a domain also known for its role in initiating Kv channel assembly and determining the compatibility of subunit interactions (35–38).
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Despite the elucidation of this general mechanism by which calcium elevation induces neuronal gene expression, a number of questions remain to be addressed. For example, there are many routes of calcium entry in neurons, and it has been widely observed that not all paths of entry are equally efficient at inducing the expression of activity-induced genes including BDNF. This observation raises the question, how is it possible that some calcium channels are tightly coupled to gene expression whereas others are not?