Hyperfunctions and theoretical physics by F.L. Pham

By F.L. Pham

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Corlicoslcroid effect in blood; 1 neutrophils; 1 lymphocytes and eosinophils T ESR; T fibrinogen, anemia 36 Rapid Review Pathology CRP; marker of necrosis and disease activily C. C-reiietive protein (CRP) 1. Acute-phase reactant 2. Clinical usefulness a. Sensitive indicatorof necrosis associated with acute inflammation • CRP is increased in inflammatory (disrupted) atherosclerotic plaques and hacterui infections. g.. rheumatoid arthritis) D. Sennn prolein electrophoresis (SPlv) in infliiinniiition (Fig.

Increase in scrum IgC 3. Table 2-4 summarizes cells involved in infiammation (Fig. 2-14A to D). TABLE 2-4. SUMMARY OF LEUKOCYTES CHARACTERISTICS Neulrophil (see Fig. 2-13) Key cell in acute inflammalion. Receptors for IgC and C^b: important in phagocytosis of opsonized bacteria. , acute myocardial infarction) Drugs inhibiting neutrophil adhesion molecules: corticosteroids, catecholamines, lithium Monocytes and macrophages (see Figs. , alveolar macrophages) Functions: phagocytosis, process antigen, enhance host immunologic response (secrele cylokines like IL-i, TNF) Causes of monocytosis: chronic inflammalion, auloimmune disease, malignancy B cells and T cells (see Figs.

B) Normal circulating pool is now part ofthe marginating pool, e. Fmdothclial cell adhesion molecules (1) Intercellular adhesion molecule (ICAM) and vascular eel! adhesion molecule (VCAM) bind to integrins on the surface of neutrophils. (2) ICAM and VCAM activation is mediated by interleukin 1 (IL-1) and tumor necrosis factor ( T N F ) . d. Leukocyte adhesion deficiency (LAID 11) Autosomal recessive disorders (2) LAD type 1 isa deficiency of C D 11 a:CI) IK, (3) LAD type 2 is a deficiency ofa selectin that binds neiilrophils.

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