Fungal Pathogenesis: Principles and Clinical Applications by Richard Calderone

By Richard Calderone

Georgetown Univ., Washington, D.C. provides learn concerned about virulence, immunity, analysis, and treatment of the most typical fungal infections. Designed for college kids, researchers, and clinicians.

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Patients are also failing antiretroviral therapies at an increasing rate [4]. These data suggest that management of cryptococcosis is highly dependent on the management of AIDS and since AIDS is still incurable, cryptococcosis in these patients remains a potential life-threatening complication. These problems do not begin to take into account cryptococcosis in underdeveloped countries, which have access to neither anti-HIV nor antifungal drugs. In these places, cryptococcosis remains as devastating as it was during the early stages of the AIDS epidemic in the United States.

Apmis 101:187–193, 1993. 107. SA Klotz, RL Smith. A fibronectin receptor on Candida albicans mediates adherence of the fungus to extracellular matrix. J Infect Dis 163:604–610, 1991. 108. E Negre, T Vogel, A Levanon, R Guy, TJ Walsh, DD Roberts. The collagen binding domain of fibronectin contains a high affinity binding site for Candida albicans. J Biol Chem 269: 22039–22045, 1994. 109. C Penn, SA Klotz. Binding of plasma fibronectin to Candida albicans occurs through the cell binding domain. Microb Pathog 17:387–393, 1994.

C. albicans bound to immobilized type IV collagen and less well to native and denatured (gelatin) type I 14 Chaffin collagen [134,144] (Table 2). Fibronectin or the peptide GRGESP were more effective inhibitors of binding to both collagens than were two RGD-containing peptides [107]. Heparin also inhibited binding of yeast cells to collagens, apparently by binding to the collagen and blocking fungal recognition [51]. The 60-kDa peptide obtained by fibronectin affinity chromatography was also obtained by collagen affinity chromatography [105].

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