By Richard Calderone
Georgetown Univ., Washington, D.C. provides learn concerned about virulence, immunity, analysis, and treatment of the most typical fungal infections. Designed for college kids, researchers, and clinicians.
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Extra resources for Fungal Pathogenesis: Principles and Clinical Applications (Mycology)
Patients are also failing antiretroviral therapies at an increasing rate . These data suggest that management of cryptococcosis is highly dependent on the management of AIDS and since AIDS is still incurable, cryptococcosis in these patients remains a potential life-threatening complication. These problems do not begin to take into account cryptococcosis in underdeveloped countries, which have access to neither anti-HIV nor antifungal drugs. In these places, cryptococcosis remains as devastating as it was during the early stages of the AIDS epidemic in the United States.
Apmis 101:187–193, 1993. 107. SA Klotz, RL Smith. A fibronectin receptor on Candida albicans mediates adherence of the fungus to extracellular matrix. J Infect Dis 163:604–610, 1991. 108. E Negre, T Vogel, A Levanon, R Guy, TJ Walsh, DD Roberts. The collagen binding domain of fibronectin contains a high affinity binding site for Candida albicans. J Biol Chem 269: 22039–22045, 1994. 109. C Penn, SA Klotz. Binding of plasma fibronectin to Candida albicans occurs through the cell binding domain. Microb Pathog 17:387–393, 1994.
C. albicans bound to immobilized type IV collagen and less well to native and denatured (gelatin) type I 14 Chaffin collagen [134,144] (Table 2). Fibronectin or the peptide GRGESP were more effective inhibitors of binding to both collagens than were two RGD-containing peptides . Heparin also inhibited binding of yeast cells to collagens, apparently by binding to the collagen and blocking fungal recognition . The 60-kDa peptide obtained by fibronectin affinity chromatography was also obtained by collagen affinity chromatography .