Diabetes Mellitus: Pathophysiology and Therapy: Bayer AG by P. Zimmet, G. Dowse, R. La Porte, C. Finch, C. Moy (auth.),

By P. Zimmet, G. Dowse, R. La Porte, C. Finch, C. Moy (auth.), Professor Dr. Werner Creutzfeldt, Professor Dr. Pierre J. Lefèbvre (eds.)

This e-book comprises the reports added on the Bayer Centenary Symposium held in Edinburg in might 1988. specialists within the box pronounced on contemporary advancements within the pathophysiology and treatment of diabetes mellitus. therefore an up to date review of diabetes mellitus examine offered to a small staff of experts is now on hand to a broader neighborhood of endocrinologists, physicians and internists.

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Additional resources for Diabetes Mellitus: Pathophysiology and Therapy: Bayer AG Centenary Symposium Edinburgh, U.K., May 25–28, 1988

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14 P. Zimmet et al. ;: 30% '0 ~ e a.. 20% 15% 10% 5% D 0% _11 (100%) 46% 27% 18% (0%) (0%) Fig. 1. Age-adjusted prevalence and native American admixture estimates of Pima Indians, San Antonio Mexican-Americans and Anglos, and Hanes II subjects. Hatched bars represent the San Antonio subpopulations [52] Epidemiological studies have provided new insights into these risk factors. " As potential risk factors for NIDDM, there appears to be a degree of heterogeneity in their effect both within and between populations [55].

Lancet II: 435-437 93. Bjorntorp P (1987) Distribution of body fat and health outcome in man. Proc Nutr Soc Aust 12: 11-22 94. Jarrett RJ (1984) Type 2 (non-insulin-dependent) diabetes mellitus and coronary heart diseasechicken, egg or neither? Diabetologia 26: 99-102 95. Zimmet P, King H (1985) The epidemiology of diabetes mellitus: recent developments. Alberti KGMM, Krall LP (eds) Diabetes annual/I. Elsevier, Amsterdam, pp 1-15 26 P. Zimmet et al. 96. Stern MP (1988) Type II diabetes mellitus.

4 (15% vs 11%). This slight difference may represent a more intensive diagnostic process among individuals with diabetic relatives. In any case, the frequency is much lower than among the siblings of type 2 diabetic patients (approximately 38% ). Identical Twins The concept that the genetic influence is much lower in type 1 than in type 2 diabetes was also supported by twin studies by Pyke and coworkers from London. In an earlier study the twins were subdivided according to the age at onset, but in a later and extended study the criterium was only the insulin dependence [3].

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